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Specifically, GHK-Cu downregulates genes associated with NF-kB activation and pro-inflammatory cytokine production while upregulating genes involved in antioxidant defense

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Although initially serving a protective role, reactive astrocytes can inadvertently exacerbate oxidative damage, disrupt neuronal function and contribute to neurodegenerative disease progression

In the central nervous system, GLP-1 RAs can either directly cross regions with a compromised blood-brain barrier or act via vagal afferent signaling to target GLP-1 receptors (GLP-1Rs) in the nucleus tractus solitarius (NTS) of the brainstem