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Journal of Clinical Epidemiology, 47, 10216

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brain ischemic disorders

With respect to NHR-49 , codon-optimized constructs for full-length NHR-49 and a fragment spanning residues 1155 were cloned into the pET-28a(+) vector (GenScript, Piscataway, NJ)

Although the study did not explain the mechanism of the changes, it did raise the possibility that this kind of change was likely the consequence of neuronal and glial cell apoptosis (Raper et al., 2018)