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Collectively, these mitochondria-targeted therapeutic strategiesranging from direct antioxidant approaches and metabolic modulation to quality control enhancement and mitochondrial transferaddress distinct but interconnected aspects of mitochondrial dysfunction in diabetic wounds (Table 2)

Despite its therapeutic potential, the clinical translation of OA is hindered by poor aqueous solubility, limited cutaneous permeation, and rapid systemic clearance, which compromise bioavailability in topical applications [13]

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Furthermore, pramlintide causes only modest mean weight loss of 1.6 kg and HbA1c reduction of 0.34Percentage compared with placebo [4]