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This study positions the BBS5/ mouse as a valuable model for BBS research and highlights GLP-1R agonists as a promising therapy for BBS-related obesity and metabolic dysfunction

Sensory information from the abdominal organs traverses the vagus nerve to the nucleus of the solitary tract in the brainstem, with projections to several other areas of the CNS, including the cerebral cortex and medulla oblongata (Liu and Forsythe, 2021)

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The goal is a gentler, steadier shift in appetite and metabolic signaling, with fewer side effects and a broader focus on overall wellbeing

The GLP-1 hormone is produced in the intestine and is considered the master control switch for appetite, cravings, and most facets of metabolism

Tuning in from Washington DC, hes a pharmacist, and economist and the Chief Science Policy Officer at the Global Healthy Living Foundation