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These changes were associated with elevated circulating levels of advanced glycation end products (AGEs), with kidney dysfunction partially mitigated by deletion of the receptor for AGEs ( Ager ) in Glp1r / mice

10.1016/S0169-328X(98)00074-6 305 WangR.BoulesM.TinerW.RichelsonE

Plochl, E., Sperl, W., Wermuth, B., and Colombo, J