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NOX1-derived ROS drive the expression of Lipocalin-2 in colonic epithelial cells in inflammatory conditions

doi:10.1007/s00405-013-2786-4 Strupp M, Dieterich M, Brandt T

These mutations in SHP2 result in hyperactive phosphatase activity, notably with elevated basal activity and result in inhibition of GH-induced insulin-like growth factor-1 (IGF-1) production (143, 151)

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For example, the chemotherapeutic agent doxorubicin generates ROS as part of its mechanism of action, contributing to its cytotoxic effects

IECs turn over completely every three to five days, making them highly dependent on efficient growth factor signaling and adequate ECM scaffolding