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Inhibition of mitochondrial (etomoxir) and peroxisomal (thioridazine) fatty acid oxidation revealed that each organelle contributes to approximately half of acetyl-CoA produced from palmitate in the DKO cells (Supplemental Figure 5G)

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The relative level of Bax was also significantly reduced in the GO@GSH-FA-treated group, further confirming the anti-apoptotic effect (Figure 11E)

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On May 9, 2024, U.S