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GLP-1RAs demonstrate anti-inflammatory properties both systemically and within the TME (for example, by modulating macrophages and T cells, and reducing NETs as described in the present review), thereby suppressing the pro-tumor inflammatory milieu (105,108-110,121)
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Today, more compounds that target the GIP receptor are emerging as a valuable complement to GLP-1 based treatments and in combination with e.g
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