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In auto-immune diseases, signaling of NF-kB is stuck in the ON position leading to cellular destruction, increased cytokine levels and more

About the Author: Julia Lloyd, MPH, RD, LDN, CDCES, is a registered dietitian and diabetes educator specializing in GLP-1 counseling

Mechanistic research in animal models and cell cultures shows that GLP-1 signaling can suppress inflammatory pathways activated by stress

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Additionally, low laminar flow found in smaller vessels promotes a quiescent state in ECs, while in larger vessels, higher physiological shear stress induces ECs' elongation and alignment parallel to the flow direction [208, 209]

Diarrhea (experienced by about 30% of users) often occurs in the early phase of treatment as digestion accelerates