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In conclusion, our study highlighted the role of -syn pathology in chronic hypoxia-induced cognitive impairment and revealed a novel underlying mechanism, providing reliable targets for future research

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The specificity of these responses relies on the spatial and temporal control of ROS production, which distinguishes transient signaling events from sustained oxidative stress

The actual mechanism is likely to occur via two sequential steps with semiquinone (SQ . ) and superoxide (O2 . ) intermediates

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