*Correspondence: Mami Noda [email protected] Present Address: Masataka Ifuku, Pharmaceutical Research Division, Takeda Pharmaceutical Company Limited, Fujisawa, Japan This article was submitted to Molecular Psychiatry, a section of the journal Frontiers in Psychiatry Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers
The researchers used antisense oligonucleotide approaches to reduce NNMT specifically in adipose tissue
Those with chronic fatigue or mitochondrial inhibitions from chemicals or toxins will have reduced function, with less bicarbonate being released and less sulfur exchange which will further reduce sulfation and glutathione formation, the very antioxidant so important to keep the mitochondria healthy
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doi: 10.1016/j.jconrel.2008.12.010
