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In dermatological contexts, the most clinically relevant gene targets include: COL1A1 and COL3A1 upregulation increases procollagen type I and III synthesis, directly counteracting age-related collagen depletion

Vascular Endothelial Growth Factor Receptor 2 (VEGFR2) Modulation Enhances VEGFR2 expression and downstream angiogenesis : BPC-157 induces rapid angiogenesis in granulation tissue via VEGFR2-associated mechanisms, promoting organized capillary formation and increased vascular density in wound regions 1-3

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