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These pancreatic effects are complemented by improved peripheral insulin responsiveness in muscle tissue and lipid storage sites, facilitating glucose disposal and reducing systemic insulin resistance - a key driver of hepatic lipid accumulation and MASH progression [28,31]

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While large-scale, untargeted metabolomic studies remain relatively uncommon, there have been small-scale attempts to prove the applicability of this strategy

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It acts as an internal defense system against free radicals and reactive oxygen species, which can damage cellular lipids, proteins and DNA