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The peptide's upregulation of VEGFR2 at both mRNA and protein levels sensitizes healing tissues to angiogenic signals while its activation of the Src-Caveolin-1-eNOS pathway generates nitric oxide essential for vascular development and tissue perfusion

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Interestingly, despite increased expression of IGF-1, the relative rate of loss of muscle mass and strength is not attenuated in virus-injected versus control muscles

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