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In experiments assessing GST metabolic stability, liver cytoplasm served as the incubation system, with 4-nitrobenzoyl chloride (PNBC) and ethacrynic acid (EA) selected as substrates and inhibitors of GST, respectively [18]

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Upon the subsequent session, a further 24% reduction would occur, amounting to approximately 2.7 pounds of fat removal, thereby facilitating an additional decrement in abdominal adiposity

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Enrolled participants had a mean baseline A1C of 8.1% in both treatment groups

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