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D) Fluo-4-loaded HEK-GLP-1R cells were challenged with either vehicle control (1% DMSO) or compound 2 (100 M) in the absence or presence of pretreatment with thapsigargin (2 M, 5 min)

This narrative review aims to synthesize current evidence on the physiological and therapeutic relevance of GLP-1 signaling in sepsis, highlight mechanistic insights from preclinical studies, and assess the translational potential of GLP-1RAs in modulating systemic inflammation, metabolic dysregulation, and organ dysfunction in septic states