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Although findings from clinical studies are promising, there are concerns that long-term exposure of GLP-1 RAs at supratherapeutic or maximum approved maintenance doses may impair muscle cell differentiation and potentially contribute to the development of sarcopenia, despite their potential to improve muscle metabolism and quality [83, 88]

S-Nitrosylation of p62 inhibits autophagic flux to promote -Synuclein secretion and spread in Parkinsons disease and Lewy body dementia