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At the cellular interface, dermal fibroblasts regulate spreading, migration, and extracellular matrix (ECM) turnover through integrin-dependent adhesion signaling (e.g., FAK/ERK pathways), which is central to fibroblastECM homeostasis [48]

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This unique characteristic has made BPC-157 a compelling tool for researchers exploring novel mechanisms of tissue repair, cytoprotection, and homeostatic regulation

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