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This finding is particularly noteworthy given that some trials extended to 24 weeks of continuous exposure and included participants with obesity-related comorbidities who might be expected to have higher baseline health risks

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If SQOR activity declines or its regulation fails, H 2 S shifts from a signaling molecule to a cytotoxin, leading to mitochondrial dysfunction, decreased ATP production, and heightened inflammation

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(145) demonstrated that Cul5 deficiency elevates levels of chemokines such as CCL2, CCL5, CXCL5, and CXCL10, which are critical for monocyte recruitment (149, 150, 155158)