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Furthermore, Cornu and colleagues found an autocrine loop where GLP-1 prevents the death of -cells in an IGF-1R-dependent manner via secretion of IGF-2 from the same cells (39)

mAbs 8 , 14561466 (2016)

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National Human Genome Research Institute

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If binding of two IGF1 molecules to the IGF1R dimer is required for receptor activation, the IGF1R activity in cells transfected with WT and F701A plasmids should be 25% of that in cells transfected with the WT plasmid alone