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MPTP-mediated ablation of enteric dopaminergic neurons, which reduced dopamine release in the gut, increased the production of inflammatory cytokines by invariant NK T-cells during hepatitis by preventing dopamine-induced repression of inflammatory signaling via the D1-like receptor-PKA pathway (Xue, Zhang et al., 2018)

The protective potency of tea polyphenols is positively correlated with the number of phenolic hydroxyl groups in their phenol rings [26]

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These activities were shown to be part of a TLR4-dependent autocrine loop by which DAT dynamically regulates the amount of dopamine in the local microenvironment to modulate immune functions such as cytokine release and phagocytosis (Mackie et al., 2022)

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