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In particular, transient interactions with residues at the base of the binding cavity correlate with enhanced kinetics for G protein activation, providing a rationale for differences in G protein-mediated signalling efficacy from distinct agonists

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For transfer across the basal membrane of the enterocyte, T + is converted into thiamine pyrophosphate (TPP) by thiamine pyrophosphokinase-1 (TPK1) and dephosphorylation of TPP to thiamine monophosphate (TMP) and thiamine by prostatic acid phosphatase (ACPP) [11]

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Non-pharmacological interventions for traumatic brain injury