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These malfunctional astrocytes are characterised by a prominent functional asthenia and lose key homoeostatic proteins, such as glutamate transporters, K ir 4.1 channels, glutamine synthetase and gap junctional protein connexin 43, which results in an uncoupling of astrocytic syncytia

The peptide distributes systemically and migrates to injury sites

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doi: 10.1007/s00264-020-04687-7

[11] If you do have pernicious anemia, then supplementation may not be the answer

SPAdes: a new genome assembly algorithm and its applications to single-cell sequencing