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Long-term, lower level exposure results in cadmium accumulation in the kidneys and thyroid gland, and can contribute to thyroid issues, kidney disease, infertility, uterine fibroids, and other reproductive tract diseases if the build-up is high enough

Within the last decade, the brain was identified as a target organ for APAP toxicity in newborn laboratory mice based on profound, long-term loss of cognitive function observed following exposure to relatively low drug doses [12]

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Liu established a mouse model of aplastic anemia with iron overload and discovered that via stimulating the Nrf2/HO-1 and PI3K/AKT/mTOR pathways, panaxadiol saponin suppressed ferroptosis in these mice (Xi et al., 2023)