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These previous studies indicated that Shh/Gli-1 signaling may represent a potential therapeutic target for the regulation of microglial activation in central nervous system diseases

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Benoist 2014, PMID 25187433, retracted 2025 per notice PMID 40312093) is potentiation of hepatocyte growth factor (HGF) signaling at the c-Met (MET) receptor, driving dendritic spine formation and synaptogenesis via PI3K-AKT

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They recruit and activate innate immune cells (such as macrophages) and promote local inflammation and tissue damage

When the majority of that literature originates from a single research group, uses the same animal model, and has not been independently replicated, the effective evidence base is narrower than the publication count implies